Publications

Mass spectrometry-based metabolomics study of nicotine exposure in THP-1 monocytes

Biologiske effekter av dentale adhesivsystemer

Digital manufacturing techniques and the in vitro biocompatibility of acrylic-based occlusal device materials

Investigation of biological effects of HEMA in 3D-organotypic co-culture models of normal and malignant oral keratinocytes

Biological aspects of modern dental composites

Luting cements in a nutshell

In Vitro Effects of Dental Monomer Exposure – Dependence on the Cell Culture Model

Ion release from three different dental alloys – effect of dynamic loading and toxicity of released elements

Establishing a macrophage model with relevance for oral methacrylate monomer exposures: Attenuated Staphylococcus aureus-induced cytokine release from human macrophages

The dental monomer HEMA causes proteome changes in human THP‐1 monocytes

The dental monomer 2-hydroxyethyl methacrylate (HEMA) causes transcriptionally regulated adaptation partially initiated by electrophilic stress

Di-n-butyl phthalate modifies PMA-induced macrophage differentiation of THP-1 monocytes via PPARγ

Presence and leaching of bisphenol a (BPA) from dental materials

Nrf2 Signaling and Cytoprotection in Human Epithelial Airway Cells Exposed to HEMA

Side effects of chemical compounds used in dental clinics

Relation between In Vitro Toxicity and the Thiol-Reactivity of Methacrylates in Resin-Based Biomaterials

The dental monomer hydroxyethyl methacrylate (HEMA) counteracts lipopolysaccharide-induced IL-1β release-Possible role of glutathione

Dentale restaureringsmaterialer: Biologiske egenskaper og deres kliniske konsekvenser

Proteome Changes Indicate Oxidative Stress and Protein Damage after Methacrylate Exposure in Human Monocyte Cell Line

Effect of Components in Resin-Based Dental Restoratives on Protein-S-Glutathionylation

Exposure to Dental Monomer Hydroxyethyl Methacrylate (HEMA) Induces Increased Transcription of Genes Involved in Oxidative Stress Responses

Toxicity of plast materials

Cell toxicity of 2-hydroxyethyl methacrylate (HEMA): the role of oxidative stress

Sub-lethal Concentrations of HEMA alter Protein Expression in Human Monocytes

TEGDMA and filler particles from dental composites additively attentuate LPS-induced cytokine release from the macrophage cell line RAW 264.7

Cell toxicity of methacrylate monomers-the role of glutathione adduct formation

Dental monomers inhibit LPS-induced cytokine release from the macrophage cell line RAW264.7

Mono-2-ethylhexylphthalate (MEHP) induces TNF-α release and macrophage differentiation through different signalling pathways in RAW264.7 cells

Biologiske effekter av dentale fyllpartikler

Metakrylat-exponering via luftvägarna

Sammendrag av doktorgradsavhandling: «Toxicity of resin based dental restorative materials in vitro»

Biotransformation enzymes and lung cell response to 2-hydroxyethyl-methacrylate

Stoffer i plastfyllingsmaterialer kan gi celleskader

DNA damage, cell-cycle arrest and apoptosis induced in BEAS-2B cells by 2-hydroxyethyl methacrylate (HEMA)

Role of thiol-complex formation in 2-hydroxyethyl-methacrylate-induced toxicity in vitro

DNA damage and DNA damage responses in THP-1 monocytes after exposure to spores of either Stachybotrys chartarum or Aspergillus versicolor or to T-2 toxin

Filler Particles Used in Dental Biomaterials Induce Production and Release of Inflammatory Mediators In Vitro

HEMA reduces cell proliferation and induces apoptosis in vitro

Dentale plastmaterialer har potensial for å utløse toksisk reaksjoner

Apoptosis induced by the monomers HEMA and TEGDMA involves formation of ROS and differential activation of the MAP-kinases p38, JNK and ERK

Involvement of NADPH Oxidase and iNOS in Rodent Pulmonary Cytokine Responses to Urban Air and Mineral Particles

Regulation of rat alveolar type 2 cell proliferation in vitro involves type II cAMP-dependent protein kinase

Pattern of cell death after in vitro exposure to GDMA, TEGDMA, HEMA and two compomer extracts

Research projects