Bergitte Olderbø

Senior Engineer

Biology MSc.

Bergitte Olderbø holds a master’s degree in molecular biology from University of Oslo. She conducts biocompatibility testing of dental biomaterials and research on components from these biomaterials, utilizing a diverse array of molecular biological analyses. Her research focuses on understanding how components influence immune responses in various cell lines. Furthermore, she supervises Bachelor and Master students who actively contribute to different research projects. 

Uhlig, S., Olderbø, B. P., Samuelsen, J. T., Uvsløkk, S., Ivanova, L., Vanderstraeten, C., Grutle, L. A. & Rangel-Huerta, O. D. (2024).
Mass spectrometry-based metabolomics study of nicotine exposure in THP-1 monocytes.
Scientific Reports, Vol. 14, Issue 1, SP 14957, SN 2045-2322.
Doi: 10.1038/s41598-024-65733-7 Lenke til et annet nettsted, åpnes i nytt vindu.

Bølling, A.K., Olderbø, B.P., Samuelsen, J.T. & Rukke, H.V. (2019).
Establishing a macrophage model with relevance for oral methacrylate monomer exposures: Attenuated Staphylococcus aureus-induced cytokine release from human macrophages.
Dental Materials, Vol. 35, Issue 10, SP-EP: e235-e248, SN 0109-5641.
Doi: 10.1016/j.dental.2019.07.005 Lenke til et annet nettsted, åpnes i nytt vindu.

Becher, R., Valen, H., Olderbø, B. P., Bølling, A. K. & Samuelsen, J. T. (2019). 
The dental monomer 2-hydroxyethyl methacrylate (HEMA) causes transcriptionally regulated adaptation partially initiated by electrophilic stress.
Dental Materials, Vol. 35, Issue 1, SP-EP: 125-134, SN 0109-5641.
Doi: 10.1016/j.dental.2018.11.008 Lenke til et annet nettsted, åpnes i nytt vindu.

Grytting, V. S., Olderbø, B. P., Holme, J. A., Samuelsen, J. T., Solhaug, A., Becher, R. & Bølling, A. K. (2019).
Di-n-butyl phthalate modifies PMA-induced macrophage differentiation of THP-1 monocytes via PPARγ.
Toxicology in Vitro, Vol. 54, SP-EP: 168-177, SN 0887-2333.
Doi: 10.1016/j.tiv.2018.09.004 Lenke til et annet nettsted, åpnes i nytt vindu.

 

Nrf2 Signaling and Cytoprotection in Human Epithelial Airway Cells Exposed to HEMA Lenke til et annet nettsted, åpnes i nytt vindu.